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HWHY Teaches us to separate from the unclean, and be set-a-part onto Him. All About Human Sperm
The House of Yahweh teach that sperm is clean to eat, but eating sperm is cannibalism!
A spermatozoon is about two thousandths of an inch long. There are about 200-500 million of them in a single ejaculation. Unlike every cell of the male body, which has 46 chromosomes (in 23 pairs), these cells have only 23 (not in pairs). The same is true for the female eggs.
Read what the devil is up to in our pets. This study is on only one of more than 35 parasites that are infectious to cat, dog, and humans. Parasite 'turns women into sex kittens'December 26, 2006 05:27pm
Article from:
AAP
About 40 per cent of the world's population is infected with Toxoplasma gondii, including about eight million Australians. Human infection generally occurs when people eat raw or undercooked meat that has cysts containing the parasite, or accidentally ingest some of the parasite's eggs excreted by an infected cat. The parasite is known to be dangerous to pregnant women as it can cause disability or abortion of the unborn child, and can also kill people whose immune systems are weakened. Until recently it was thought to be an insignificant disease in healthy people, Sydney University of Technology infectious disease researcher Nicky Boulter said, but new research has revealed its mind-altering properties. "Interestingly, the effect of infection is different between men and women,'' Dr Boulter writes in the latest issue of Australasian Science magazine. "Infected men have lower IQs, achieve a lower level of education and have shorter attention spans. They are also more likely to break rules and take risks, be more independent, more anti-social, suspicious, jealous and morose, and are deemed less attractive to women. "On the other hand, infected women tend to be more outgoing, friendly, more promiscuous, and are considered more attractive to men compared with non-infected controls. "In short, it can make men behave like alley cats and women behave like sex kittens''. Dr Boulter said the recent Czech Republic research was not conclusive, but was backed up by animal studies that found infection also changes the behaviour of mice. The mice were more likely to take risks that increased their chance of being eaten by cats, which would allow the parasite to continue its life cycle. Rodents treated with drugs that killed the parasites reversed their behaviour, Dr Boulter said. Another study showed people who were infected but not showing symptoms were 2.7 times more likely than uninfected people to be involved in a car accident as a driver or pedestrian, while other research has linked the parasite to higher incidences of schizophrenia. "The increasing body of evidence connecting Toxoplasma infection with changes in personality and mental state, combined with the extremely high incidence of human infection in both developing and developed countries, warrants increased government funding and research, in particular to find safe and effective treatments or vaccines,'' Dr Boulter said.
T. gondii constructing daughter scaffolds within the mother cell.
Mind Control by Parasites
By Bill Christensen posted: 10 February 2006 06:41 am ET http://www.livescience.com/technology/060210_technovelgy.html this article is from this website, I rather you follow the link to read first hand. Half of the world's human population is infected with Toxoplasma, parasites in the body—and the brain. Remember that. Toxoplasma gondii is a common parasite found in the guts of cats; it sheds eggs that are picked up by rats and other animals that are eaten by cats. Toxoplasma forms cysts in the bodies of the intermediate rat hosts, including in the brain. Since cats don't want to eat dead, decaying prey, Toxoplasma takes the evolutionarily sound course of being a "good" parasite, leaving the rats perfectly healthy. Or are they? Oxford scientists discovered that the minds of the infected rats have been subtly altered. In a series of experiments, they demonstrated that healthy rats will prudently avoid areas that have been doused with cat urine. In fact, when scientists test anti-anxiety drugs on rats, they use a whiff of cat urine to induce neurochemical panic. However, it turns out that Toxoplasma-ridden rats show no such reaction. In fact, some of the infected rats actually seek out the cat urine-marked areas again and again. The parasite alters the mind (and thus the behavior) of the rat for its own benefit. If the parasite can alter rat behavior, does it have any effect on humans? Dr. E. Fuller Torrey (Associate Director for Laboratory Research at the Stanley Medical Research Institute) noticed links between Toxoplasma and schizophrenia in human beings, approximately three billion of whom are infected with T. gondii: · Toxoplasma infection is associated with damage to astrocytes, glial cells which surround and support neurons. Schizophrenia is also associated with damage to astrocytes. · Pregnant women with high levels of antibodies to Toxoplasma are more likely to give birth to children who will develop schizophrenia. · Human cells raised in petri dishes, and infected with Toxoplasma, will respond to drugs like haloperidol; the growth of the parasite stops. Haloperidol is an antipsychotic, used to treat schizophrenia. Dr. Torrey got together with the Oxford scientists, to see if anything could be done about those parasite-controlled rats that were driven to hang around cat urine-soaked corners (waiting for cats). According to a recent press release, haloperidol restores the rat's healthy fear of cat urine. In fact, antipsychotic drugs were as effective as pyrimethamine, a drug that specifically eliminates Toxoplasma. Are parasites like Toxoplasma subtly altering human behavior? As it turns out, science fiction writers have been thinking about whether or not parasites could alter a human being's behavior, or even take control of a person. In his 1951 novel The Puppet Masters, Robert Heinlein wrote about alien parasites the size of dinner plates that took control of the minds of their hosts, flooding their brains with neurochemicals. In this excerpt, a volunteer strapped to a chair allows a parasite to be introduced; the parasite rides him, taking over his mind. Under these conditions, it is possible to interview the parasite; however, it refuses to answer until zapped with a cattle prod. He reached past my shoulders with a rod. I felt a shocking, unbearable pain. The room blacked out as if a switch had been thrown.. I was split apart by it; for the moment I was masterless. The pain left, leaving only its searing memory behind. Before I could speak, or even think coherently for myself, the splitting away had ended and I was again safe in the arms of my master... The panic that possessed me washed away; I was again filled with an unworried sense of well being...
"What are you?" Still not sure that parasites can manipulate the behavior of host organisms? Consider these other cases: · The lancet fluke Dicrocoelium dendriticum forces its ant host to attach to the tips of grass blades, the easier to be eaten. The fluke needs to get into the gut of a grazing animal to complete its life cycle. · The fluke Euhaplorchis californiensis causes fish to shimmy and jump so wading birds will grab them and eat them, for the same reason. · Hairworms, which live inside grasshoppers, sabotage the grasshopper's central nervous system, forcing them to jump into pools of water, drowning themselves. Hairworms then swim away from their hapless hosts to continue their life cycle. Not all science-fictional parasites are harmful; read about the Crosswell tapeworm from Brian Aldiss' 1969 story Super-Toys Last All Summer Long (the basis for the Kubrick/Spielberg film AI), which keeps people who overeat from becoming obese. Not to mention robots based on parasites. Read press release on evidence for link between Toxoplasma and schizophrenia, Suicidal grasshoppers. Story via blogger Carl Zimmer and his readers.
http://images.google.com/imgres?imgurl=http://journals.cambridge.org/fulltext_content/ERM/ERM3_01/ Expert Reviews in Molecular
Medicine:
http://www-ermm.cbcu.cam.ac.uk Ultrastructure of a Toxoplasma gondii tachyzoite James W. Ajioka, Jennifer M. Fitzpatrick and Christopher P. Reitter
Figure 1. Ultrastructure of a Toxoplasma gondii tachyzoite. The conoid defines the apical end of the parasite and is thought to be associated with the penetration of the host cell. Micronemes, rhoptries and dense granules are the three major secretory organelles, found predominately at the apical end of the parasite. Microneme proteins are released very early in the invasion process, facilitating host-cell binding and gliding motility. Rhoptry proteins are also released during invasion, and can be detected within the lumen and membrane of the newly generated parasitophorous vacuole (PV). Dense-granule proteins are released during and after the formation of the PV, modifying the PV environment for intracellular survival and replication of the parasite. The apicoplast is a plastid-like four-membrane organelle containing a 35 kb circular DNA. Most of the proteins functioning within the organelle are encoded by the nucleus, and are specifically targeted to the apicoplast. This targeting involves the secretory pathway, including the rough endoplasmic reticulum (ER) and a Golgi body situated immediately apical to the nucleus. Targeted proteins have a bipartite N-terminal extension, consisting of an ER signal sequence followed by a plastid transit peptide. T. gondii cells have a single nucleus and a single mitochondrion. It is hypothesised that reliance on the mitochondrion for cellular metabolism differs according to the life-cycle stage of the parasite (fig001jac). Toxoplasma gondii
Parasite Primer
From
Cathy Wong, About.com Health's Disease and Condition content is reviewed by our Medical Review Board http://altmedicine.about.com/cs/conditionsitoq/a/ParasitePrimer.htm
Symptoms and Sources of Infection of Different Parasites
Learn
about the various types of parasites, symptoms, and the sources of infection and
spread. · Giardia lamblia originates in infected humans and animals. It is transmitted through water, especially in the Rocky mountains, food, and contact with feces often due to poor hygiene and handwashing. Giardia infects the small intestines. It can take up to three weeks before symptoms of giardia appear. Giardia can result in symptomless intestinal infection. It can also produce watery diarrhea, foul-smelling stools, nausea, stomach cramps, bloating, gas, low energy, and weight loss. Multiple stool samples are often necessary for diagnosis. · Cryptosporidium parvum is often transmitted by contact with human feces that contains infectious cysts, for example poor handwashing. Cryptosporidium parvum is transmitted in water. It is the most prevalent waterbourne parasite in the United States. In people with healthy immune systems, it can be symptomless or symptoms can include watery diarrhea that lasts about 10 days, nausea, cramps, and fever. In people who are immunocompromised, there can be severe diarrhea with weakness, weight loss, which can be life-threatening. · Cyclospora species is mainly seen in travelers. Recent outbreaks have been associated with contaminated water sources, such as Central American raspberries and basil. It is transmitted through contact with feces. Symptoms are similar to giardiasis. Symptoms come and go and can include diarrhea, frequent watery stools, weight loss, fatigue, bloating, or symptomless gallbladder disease. · Entamoeba histolytica is spread through water or food. Insects can carry the cysts, such as flies and cockroaches. It may take up to three months after infection before symptoms appear. It can spread through the digestive tract and travel to other organs. Most often, the infected person is a symptomless carrier. It can cause stomach pain, bloating, and diarrhea. If there is tissue destruction in the large intestines, there can be low grade fever with bloddy diarrhea. · Toxoplasma gondii is an infection that generally comes from cats. Another source of toxoplasma is undercooked meat. Symptoms include flu-like symptoms such as fever, headache, swollen lymph nodes, and fatigue. · Trichomonas vaginalis is transmitted through sexual contact or from contaminated toilet seats, towels, or bathwater. There are often no symptoms. It can cause vaginal discharge, yeast infections, and painful urination in women. In men it can cause an enlarged prostate gland and urinary inflammation.
2.
Roundworms and Hookworms · Roundworm (ascaris lumbricoides) is symptomless in many people. It is estimated that over one billion people in the world may be infected with roundworm. The source of infection is contamination of soil and vegetables with feces. Adult roundworms live in the small intestines and can exit through the mouth or nose of the infected person. Occasionally, there is obstruction of the pancreatic or bile duct, appendix, or small intestines. Dry cough, fever, and sleep disturbance may occur. Diagnosis is by stool exam for eggs and blood test. · Necator americanus (hookworms) is transmitted through unbroken skin by walking barefoot. Hookworms travel into blood and through the lung and intestines. Hookworm infection is usually symptomless. There may be itching at the area of skin penetration. There can be digestive symptoms. The worms attach to and suck the blood from the mucous of the small intestines, leading to iron deficiency anemia, low energy, and peptic ulcer-like symptoms in severe infections. · Enterobius vermicularis (pinworm) infection is common in the United States. It is transmitted through contaminated food and water. The worms live in the intestines near the rectum and travel at night outside to the skin around the anus. From there it can be transmitted through person to person contact. It can be symptomless. There is often itching at night around the anus. There can also be unusual symptoms such as hyperactivity, vision problems, vaginitis, and psychological disturbances. Tape is often applied to the anal area at night. When the tape is removed, adult worms may be seen with the unaided eye. At least 5 to 7 tests are required to rule out infection. · Trichuris trichiura (whipworm) is a large intestine parasite that rarely shows symptoms. It is transmitted by ingestion of the eggs in soil or on vegetables. Symptoms of heavy infection include diarrhea, stomach pain, rectal prolapse, and stunted growth. Updated: July 26, 2004 Important disclaimer information about this About site. Related Articles Parasitic Opportunistic InfectionsQ fever - earlySymptoms HIV - HIV /AIDS Symptoms - What are the Sympto...Whipworm infectionRotavirus http://www.medindia.net/news/view_news_main.asp?x=17133
Findings on Dangerous Parasite, Toxoplasma Gondii Published
Toxoplasma gondiiFrom Wikipedia, the free encyclopedia
Toxoplasma gondii is a species of parasitic protozoa in the genus Toxoplasma.[1] The definitive host of T. gondii is the cat, but the parasite can be carried by the vast majority of warm-blooded animals, including humans. The causative agent of Toxoplasmosis, the disease is usually minor and self-limiting but can have serious or even fatal effects on a fetus whose mother first contracts the disease during pregnancy or on an immunocompromised human or cat. Life cycleThe life cycle of T. gondii has two phases. The sexual part of the life cycle (coccidia like) takes place only in members of the Felidae family (domestic and wild cats), which makes these animals the parasite's primary host. The asexual part of the life cycle can take place in any warm-blooded animal, like other mammals (including felines) and birds. In the intermediate hosts (including felines), the parasite invades cells, forming intracellular so-called parasitophorous vacuoles containing bradyzoites, the slowly replicating form of the parasite.[2]. Vacuoles form tissue cysts mainly within the muscles and brain. Since they are within cells, the host's immune system does not detect these cysts. Resistance to antibiotics varies, but the cysts are very difficult to eradicate entirely. Within these vacuoles T. gondii propagates by a series of binary fissions until the infected cell eventually bursts and tachyzoites are released. Tachyzoites are the motile, asexually reproducing form of the parasite. Unlike the bradyzoites, the free tachyzoites are usually efficiently cleared by the host's immune response, although some manage to infect cells and form bradyzoites, thus maintaining the infection. Tissue cysts are ingested by a cat (e.g., by feeding on an infected mouse). The cysts survive passage through the stomach of the cat and the parasites infect epithelial cells of the small intestine where they undergo sexual reproduction and oocyst formation. Oocysts are shed with the feces. Animals and humans that ingest oocysts (e.g., by eating unwashed vegetables etc.) or tissue cysts in improperly cooked meat become infected. The parasite enters macrophages in the intestinal lining and is distributed via the blood stream throughout the body. Acute stage toxoplasma infections can be asymptomatic, but often gives flu-like symptoms in the early acute stages, and like flu can become, in very rare cases, fatal. The acute stage fades in a few days to months, leading to the latent stage. Latent infection is normally asymptomatic; however, in the case of immunocompromised patients (such as those infected with HIV or transplant recipients on immunosuppressive therapy), toxoplasmosis can develop. The most notable manifestation of toxoplasmosis in immunocompromised patients is toxoplasmic encephalitis, which can be deadly. If infection with T. gondii occurs for the first time during pregnancy, the parasite can cross the placenta, possibly leading to hydrocephalus, intracranial calcification, and chorioretinitis, with the possibility of spontaneous abortion or intrauterine death. What is toxoplasmosis?A single-celled parasite called Toxoplasma gondii causes a disease known as toxoplasmosis. While the parasite is found throughout the world, more than 60 million people in the United States may be infected with the Toxoplasma parasite. Of those who are infected, very few have symptoms because a healthy person's immune system usually keeps the parasite from causing illness. However, pregnant women and individuals who have compromised immune systems should be cautious; for them, a Toxoplasma infection could cause serious health problems.
How do people get toxoplasmosis?A Toxoplasma infection occurs by:
What are the symptoms of toxoplasmosis?Symptoms of the infection vary.
Lifespan
Sperm swim in and are nourished in the semen. Once inside the fallopian tube(s) they have a life span of about 5 days. This means they can be in the fallopian tube for 5 days in advance of ovulation and still fertilize the egg. The egg, unfertilized, has a life span of only about 24 hours. In the vagina, they die in only a few hours. In the open air, they die rapidly.
If there are enough sperms. If the sample has less than 20 million sperm per ml, this is considered to be a low sperm count. Less than 10 million is very low. The technical term for this is oligospermia (oligo means few). Some men will have no sperms at all and are said to be azoospermic. This can come as a rude shock because the semen in these patients look absolutely normal - it is only on microscopic examination that the problem is detected. Whether the sperms are moving well or not (sperm motility). The quality of the sperm is often more significant than the count. Sperm motility is the ability to move. Sperm are of 2 types � those which swim, and those which don�t. Remember that only those sperm which move forward fast are able to swim up to the egg and fertilise it � the others are of little use. Motility is graded from a to d, according to the World Health Organisation ( WHO) Manual criteria , as follows. Grade a ( fast progressive) sperms are those which swim forward fast in a straight line - like guided missiles. Grade b ( slow progressive) sperms swim forward, but either in a curved or crooked line, or slowly (slow linear or non linear motility) . Grade c ( nonprogressive) sperms move their tails, but do not move forward ( local motility only). Grade d ( immotile ) sperms do not move at all . Sperms of grade c and d are considered poor. Why do we worry about poor motility ? If motility is poor, this suggests that the testis is producing poor quality sperm and is not functioning properly � and this may mean that even the apparently motile sperm may not be able to fertilise the egg. Whether the sperms are normally shaped or not - what is called their form or morphology. Ideally, a good sperm should have a regular oval head, with a connecting mid-piece and a long straight tail. If too many sperms are abnormally shaped (round heads; pin heads; very large heads; double heads; absent tails) this may mean the sperm are abnormal and will not be able to fertilise the egg. Many labs use Kruger "strict " criteria ( developed in South Africa ) for judging sperm normality. Only sperm which are "perfect" are considered to be normal. A normal sample should have at least 15% normal forms ( which means even upto 85% abnormal forms is considered to be acceptable !) Sperm clumping or agglutination. Under the microscope, this is seen as the sperms sticking together to one another in bunches. This impairs sperm motility and prevents the sperms from swimming upto through the cervix towards the egg. Putting it all together, one looks for the total number of "good" sperms in the sample - the product of the total count, the progressively motile sperm and the normally shaped sperm. This gives the progressively motile normal sperm count which is a crude index of the fertility potential of the sperm. Thus, for example, if a man has a total count of 40 million sperm per ml; of which 40% are progressively motile; and 60% are normally shaped; then his progressively motile normal sperm count is : 40 X 0.40 X 0.60 = 9.6 million sperm per ml. If the volume of the ejaculate is 3 ml, then the total motile sperm count in the entire sample is 9.6 X 3 = 28.8 million sperm. Whether pus cells are present or not. While a few white blood cells in the semen is normal, many pus cells suggests the presence of seminal infection. Some labs use a computer to do the semen analysis. This is called CASA, or computer assisted semen analysis. While it may appear to be more reliable ( because the test has been done "objectively" by a computer), there are still many controversies about its real value, since many of the technical details have not been standardised, and vary from lab to lab. A normal sperm report is reassuring, and usually does not need to be repeated. If the semen analysis is normal, most doctors will not even need to examine the man, since this is then superfluous. However, remember that just because the sperm count and motility are in the normal range, this does not necessarily mean that the man is "fertile". Even if the sperm display normal motility, this does not always mean that they are capable of "working" and fertilising the egg. The only foolproof way of proving whether the sperm work is by doing IVF ( in vitro fertilisation)! Poor sperm tests can results from:
If the sperm test is abnormal, this will need to be repeated 3-4 times over a period of 3-6 months to confirm whether the abnormality is persistent or not. Don�t jump to a conclusion based on just one report - remember that sperm counts do tend to vary on their own! It takes six weeks for the testes to produce new sperm - which is why you need to wait before repeating the test. It also makes sense to repeat it from another laboratory, to ensure that the report is valid. What if the sperm count is persistently poor ? Then other tests may be advised, to try to pinpoint what the problem is; and these are described in the next chapter. |
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Sperm, is short for 
